Protein kinase C is one of the most important enzymes in Alzheimer’s disease, but also one of the trickiest. This enzyme maintains the balance between cell growth and cell death (review of protein kinase C). The over-activation of protein kinase C during the early stages of Alzheimer’s disease leads to mitochondrial dysfunction, inflammation, deficits in neurotransmitters needed for the retrieval of short-term memory, balanced mood, sleep, social recognition, and alertness, inflammation, the lack of regeneration of neurons and synapses in the hippocampus, and the death of neurons and synapses. The decline in protein